calcium glu.pdf - ACTIVE LEARNING TEMPLATE Medication jeniell forbes STUDENT NAME Calcium Gluconate MEDICATION REVIEW MODULE CHAPTER and electrolyte

calcium glu.pdf - ACTIVE LEARNING TEMPLATE Medication...

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Unformatted text preview: ACTIVE LEARNING TEMPLATE: Medication jeniell forbes STUDENT NAME______________________________________ Calcium Gluconate MEDICATION___________________________________________________________________________ REVIEW MODULE CHAPTER____________ and electrolyte replacements/supplements CATEGORY CLASS__mineral _____________________________________________________________________ PURPOSE OF MEDICATION Expected Pharmacological Action Essential for nervous, muscular, and skeletal systems. Maintain cell membrane and capillary permeability. Act as an activator in the transmission of nerve impulses and contraction of cardiac, skeletal, and smooth muscle. Essential for bone formation and blood coagulation. Therapeutic Use Emergency treatment of hyperkalemia and hyper-magnesemia and adjunct in cardiac arrest or calcium channel blocking agent toxicity(calcium chloride, calcium gluconate).Replacement of calcium indeficiency states Complications May cause vasodilation with resulting hypotension, bradycardia, arrhythmias, and cardiac arrest. Transient increases in BP may occur during IV administration, especially in geriatric patients or in patients with hypertension Medication Administration iV (Adults):500–800 mg (maximum 3 g/dose).IV (Children, Infants, and Neonates):60–100 mg/kg/dose (maximum 3 g/dose). Contraindications/Precautions Contraindicated in: Hypercalcemia; Renal calculi; Ventricular fibrillation. Use Cautiously in:Patients receiving digitalis glycosides; Severe respiratory in-sufficiency; Renal disease; Cardiac disease. Interactions Drug-Drug: Hypercalcemia increases the risk of digoxintoxicity. Chronic use with antacids in renal insufficiency may lead to milk-alkali syndrome. Ingestion by mouth decreases the absorption of orally administered tetracyclines,fluoroquin-olones,phenytoin, andiron salts. Excessive amounts may decrease the effects of calcium channel blockers. Decreases absorption of etidronate and risedron-ate(do not take within 2 hr of calcium supplements). May decrease the effectiveness of atenolol. Concurrent use with diuretics (thiazide)may result in hypercalcemia. May decrease the ability of sodium polystyrene sulfonateto decrease serum potassium. Drug-Food:Cereals,spinach,orrhubarbmay decrease the absorption of calcium supplements. Calcium acetate should not be given concurrently with other calcium supplements. Nursing Interventions Monitor BP, pulse, and ECG frequently throughout parenteral therapy. Administer slowly. High concentrations may cause cardiac arrest. Rapid administration may cause tingling, sensation of warmth, and a metallic taste. Halt infusion if these symptoms occur, and resume infusion at a slower rate when they subside Client Education Evaluation of Medication Effectiveness Increase in serum calcium levels. Decrease in the signs and symptoms of hypocalcemia. Decrease in the signs and symptoms of Magnesium toxcity ACTIVE LEARNING TEMPLATES Encourage patients to maintain a diet adequate in vitamin. Do not administer concurrently with foods containing large amounts of oxalicacid(spinach, rhubarb), phytic acid (brans, cereals), or phosphorus (milk or dairyproducts). Administration with milk products may lead to milk-alkali syndrome(nausea, vomiting, confusion, headache). Do not take within 1– 2 hr of other medications if possible.●Instruct patients on a regular schedule to take missed doses as soon as possible,then go back to regular schedule.●Advise patient to avoid excessive use of tobacco or beverages containing alcohol or caffeine Therapeutic Procedure  A7 ...
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