PLP_405_Lecture_10 - pathogens produce haustoria 6 What...

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PLP 405 Lecture 10 Review and Discussion Questions 1. What is the composition of the primary cell wall and the middle lamella? How do fungi penetrate through these materials? 2. What are the different types of cell wall degrading enzymes produced by fungi and bacteria? What are the roles of these enzymes in pathogenesis? 3. How do endo-acting pectinases differ from exo-acting pectinases (i.e. the difference between endopolygalacturonase and exopolygalacturonase)? Which of these types of pectinases are most likely to be involved in allowing pathogens to move through cell walls? 4. What is tissue maceration? How does this occur? 5. What are haustoria? What is the function of these structures? What types of
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Unformatted text preview: pathogens produce haustoria? 6. What role do pathogen produced toxins play in pathogenesis? How do host-specific toxins differ from non-host-specific toxins? 7. Some toxins are known to be required for pathogenicity and are known as pathogenicity factors while others enhance the ability of the pathogen to cause disease (increase virulence) and are thus know as virulence factors. 8. Are cell wall degrading enzymes generally considered to be virulence or pathogenicity factors? 9. What other types of pathogen genes are required for pathogenicity? How do some pathogens deal with preformed antimicrobial compounds?...
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This note was uploaded on 03/19/2008 for the course PLP 405 taught by Professor Hammerschmidt during the Spring '08 term at Michigan State University.

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