Wilson's Disease.docx - Topic Wilsons Disease Student Name Course Submission Date October 9th 2017 Key terms Copper storage disease Hepatolenticular

Wilson's Disease.docx - Topic Wilsons Disease Student Name...

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Topic: Wilson’s Disease Student Name: Course: Submission Date: October 9 th 2017 Key terms: Copper storage disease Hepatolenticular degeneration
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INTRODUCTION Definition Wilson’s disease is an infrequent genetic disorder categorized by excessive copper deposits in the liver, brain, and other body tissues [Ric16] . Surplus of copper in the liver is toxic and if not detected early is terminal. Copper ingested is normally absorbed by the small intestine and circulated by the blood to the liver for storage with any extra copper excreted by the bile. When the biliary excretion isn’t working, copper build-up develops hence causing Wilson’s disease. It’s named after Samuel Kinnear Wilson who pinpointed it in 1912 and is known to affect one out of every thirty thousand people in the world. Wilson’s disease is also known as Copper storage disease and Hepatolenticular degeneration Symptoms Wilson’s disease exists from birth and manifests itself between the age of six and forty years after copper deposits have build-up in the liver, eyes, brain or other body organ. It’s signs and symptoms differ in response to the affected body organ. Symptoms vary from yellow pigmentation on the skin, whiteness in the eyes (jaundice)and liver dysfunction [Ban15] . Wilson’s disease displays through three major forms as follows; Chronic active hepatitis Cirrhosis Fulminant hepatic failure Some of the early indicators of Wilson’s disease includes; Trembling on the arms and legs Trouble talking Too much saliva Clumsiness Change in body posture and movement Other signs at a progressive or later stage include;
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