IGA9e_ch15_labels - 070207

IGA9e_ch15_labels - 070207 - Figure 15-10 Unnumbered figure...

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CHAPTER 15 Mutation, Repair, and Recombination Copyright 2008 © W H Freeman and Company
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CHAPTER OUTLINE 15.1 The phenotypic consequences of DNA mutations 15.2 The molecular basis of spontaneous mutations 15.3 The molecular basis of induced mutations 15.6 Cancer: an important phenotypic consequence of mutation
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Consequences of point mutations within genes Figure 15-2
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Consequences of point mutations within genes Figure 15-2 part 1
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Consequences of point mutations within genes Figure 15-2 part 2
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Consequences of point mutations within genes Figure 15-2 part 3
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Unnumbered figure pg 515
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Point mutations can alter mRNA splicing Figure 15-3
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Consequences of point mutations on gene products Figure 15-4
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“Fluctuation test” hypotheses Figure 15-5
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Replica plating Figure 15-6
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Replica plating demonstrates the presence of mutants before selection Figure 15-7
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Bases may take on rare tautomeric forms prone to mismatch Figure 15-8a
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Bases may take on rare tautomeric forms prone to mismatch Figure 15-8b
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Tautomeric shifts in DNA bases may cause mutations Figure 15-9
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Indel mutations result in frameshifts
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Unformatted text preview: Figure 15-10 Unnumbered figure pg 522 Unnumbered figure pg 523 Oxygen radicals can damage DNA Figure 15-11 Trinucleotide repeats in the FMR-1 gene hinder transcription Figure 15-12 Slipped mispairing causes repeat expansion Figure 15-13 Alternative pairings for 5-bromouracil (5-BU) Figure 15-14 Alternative pairings for 2-aminopurine Figure 15-15 Unnumbered figure pg 527 Alkylation-induced specific mispairings Figure 15-16 Intercalating agents Figure 15-17 Intercalating agents Figure 15-17a Intercalating agents Figure 15-17b UV-light-generated photoproducts Figure 15-18 Aflatoxin B1 forms a bulky addition product Figure 15-19 The Ames test reveals mutagenic compounds Figure 15-20 Ames test of aflatoxin B 1 mutagenicity Figure 15-21 Normal cells and cells transformed by an oncogene Figure 15-35 The ras oncogene is continuously active Figure 15-36 The ras oncogene is continuously active Figure 15-36a The ras oncogene is continuously active Figure 15-36b Table 15-1...
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