NTD 414 Cirrhosis (1).docx - Kanter Keller 1 Liver Cirrhosis A Case Study Approach Maggie Kanter Alexis Keller NTD 414 Introduction Kanter Keller 2

NTD 414 Cirrhosis (1).docx - Kanter Keller 1 Liver...

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Kanter, Keller 1 Liver Cirrhosis A Case Study Approach Maggie Kanter, Alexis Keller NTD 414 Introduction:
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Kanter, Keller 2 Affecting approximately 130 to 150 million people worldwide each year (Krucik, 2014), Hepatitis C has cultivated into a universal pandemic since its first appearance in 1989. According to the CDC about 3.2 million people in the United States alone are living with chronic Hepatitis C. (Krucik, 2014). This condition is transmitted through blood or bodily fluid exposure through someone who is infected with the strain, HCV. During this time, our liver is attacked by the rapidly growing infectious strain, which later causes inflammation that typically does not subside. Inflammation causes permanent fibrous damage to the healthy tissue of the liver. Approximately 55-85 percent of Hepatitis C patients will develop a chronic HCV infection (Krucik, 2014). No vaccines or cures have been discovered to rid of the HCV strain, but treatments are available, and necessary for patients with this disease. Although signs and symptoms may initially be silent, there are things things to look out for if there is concern. Symptoms like fatigue, sore muscles, nausea or poor appetite, dark urine and jaundice (Merck,2017). If this condition goes untreated, the persistent inflammation from the infectious strain will permanently damage the healthy tissue of the liver and replace itself with scarred tissue, also known as Cirrhosis. Etiology: The human body only requires about 10 to 20 percent of liver function, but with persistent damage and scarring, cirrhosis can impair most if not all liver function when gone untreated. The liver being the largest organ in our body, its functionality is essential to our quality of life. When its toxin and bacteria cleansing capabilities are impaired, our quality of life declines. The liver is responsible for the conversion and storage of glucose from galactose and fructose, bile production and secretion, the distribution of digested nutrients via portal vein, the storage of iron, the conversion of ammonia to urea, the clearance of bilirubin, and clearing the blood of harmful
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Kanter, Keller 3 drugs, toxins, and substances. The Damage and scarring from cirrhosis causes the liver to be unable to perform its normal functions. Clinical manifestations of cirrhosis include: enlarged liver from necrosis, ascites, edema, elevated AST, reduced BSP clearing time, vitamin deficiencies, decreased hematocrit and hemoglobin, jaundice, lack of appetite, delirium, fever, gallstones, ulcers, GERD, gastritis, diarrhea, portal hypertension, hepatic encephalopathy. Cirrhosis is a progressive chronic condition that mainly evolves from chronic viral Hepatitis or alcoholic hepatitis. The difference between viral Hepatitis and alcoholic Hepatitis is that alcoholic hepatitis can manifest into hepatic encephalopathy and is lifestyle induced. As a result of alcohol abuse and poor diet choices, toxins remain stagnant in the bloodstream causing permanent damage to the liver (Vienken,2006). In contrast, viral hepatitis is caused by a viral infection that can be passed from person to person.
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  • Spring '17
  • Liver cirrhosis

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