NR 281 Unit 3 ROK Pulmonary and Skin.docx

NR 281 Unit 3 ROK Pulmonary and Skin.docx

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Atlanta Campus Review of Knowledge Review of Knowledge (ROK) Course NR 283 Exam 2 Skin and Pulmonary Purpose The Review of Knowledge (ROK) focuses on course outcomes, unit outcomes and key concepts to enhance student success on exams. Use this document as a focused guide for study in conjunction with assigned readings, PowerPoint presentations, and other course resources. Supplement this document by outlining your own notes from the textbook chapters and place your expanded more detailed notes in the Student Notes column. This document should NOT be used as a substitute for completing the assigned readings. Key Concepts Chapters Student Notes Pulmonary Stress and Coping: Infections Pneumonia Types Community Acquired Bacterial Hospital Acquired 26 Pneumonia- causes an accumulation of fluid collection in the alveoli. Can be seen on an x-ray. Community acquired- Streptococcus pneumonia- The most common cause of bacterial pneumonia is Streptococcus pneumonia. This type of pneumonia can occur on its own or after you have had a cold or the flu . May affect one part (lobe) of the lung, called lobar pneumonia. Mycoplasma pneumonia- Milder symptoms- often called walking pneumonia Hospital Acquires or Ventilation acquired (VAT)- Pneumonia acquired while staying in the hospital. Patients on ventilators are at higher risk for developing VAT. Pseudomonas aerginosa Staphylococcus aureus Weakened or suppressed immune system. People who have HIV/AIDS, who’ve had organ transplant, or who POET - ROK Form 7.7.16 1
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Atlanta Campus Review of Knowledge Key Concepts Chapters Student Notes Immune Compromised Etiology Viral Influenza Pathophysiologic Process Clinical manifestations and complications Diagnostics receive chemo or on long term steroids are at risk Viral-some viruses that cause colds and the flu can cause pneumonia. Viruses are the most common cause of pneumonia in children under 5 years of age. It is usually mild, however, some cases can be very serious. The invading organism causes symptoms, in part, by provoking an overly exuberant immune response in the lungs. The small blood vessels in the lungs ( capillaries ) become leaky, and protein- rich fluid seeps into the alveoli. This results in a less functional area for oxygen-carbon dioxide exchange. The patient becomes relatively oxygen deprived, while retaining potentially damaging carbon dioxide. The patient breathes faster and faster, in an effort to bring in more oxygen and blow off more carbon dioxide. Mucus production is increased, and the leaky capillaries may tinge the mucus with blood. Mucus plugs actually further decrease the efficiency of gas exchange in the lung. The alveoli fill further with fluid and debris from the large number of white blood cells being produced to fight the infection.
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