HUN4446 hepatitis - • secondary • potential causes •...

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more rapid progression to “normal” diet dietary adjustments for: diarrhea constipation flatus odor Hepatic Steatosis: Fatty Liver Fatty Liver liver synthesizes too much fat (increased TG synthesis) liver oxidizes too little fat (decreased beta oxidation) liver takes up too much fat from blood (released by AT cells) liver releases too little fat back into the blood (TG trapped in the liver) combination of above early sign of liver deterioration categories: nonalcoholic fatty liver (NAFLD) primary secondary alcoholic fatty liver fatty liver due to pregnancy Nonalcoholic fatty liver disease (NAFLD) primary: associated with insulin resistance diabetes, obesity-often present presentation: asymptomatic--> NASH--> cirrhosis--> liver failure dx: evidence of fatty liver, no hx excessive alcohol consumption tx: wt loss, exercise
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Unformatted text preview: • secondary: • potential causes: • nutritional: rapid wt loss, kwashiokor/staration, TPN, intestinal bypass surgery • drugs • certain diseases: HIV infection, inflammatory bowel dz • tx: eliminate cause Hepatitis ➢ inflammation of the liver due to : • viruses • bacteria • obstruction • toxins • fat deposition (alcoholic or non alcoholic fatty liver) • drugs ➢ Acute Viral Hepatitis: • Types of viral hepatitis • Hep A: fecal-oral route via contaminated food or drinking water (vaccine) • Hep B: serum hepatitis (vaccine) • Hep C: blood to blood contact (NO vaccine) • asymptomatic • human disease • Hep D: needs Hep. B virus to spread • Hep E: fecal- oral route • clinical manifestations • jaundice • dark urine • anorexia • fatigue • headache...
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This note was uploaded on 10/25/2009 for the course HUN 4446 taught by Professor Mathews during the Spring '09 term at University of Florida.

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HUN4446 hepatitis - • secondary • potential causes •...

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