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Unformatted text preview: gallstones;biliary tract disease alcohol other causes: surgery/trauma cystic fibrosis certain mutations of CF gene (even without development of CF) other: hypertriglyceridemia penetrating duodenal ulcer some medications cancer viral infection acute pancreatitis proposed pathophysiology: primary acinar cell injury or damage to pancreas or blockage of sphincter of Oddi retention of pancreatic enzymes premature activation of pancreatic enzymes recruitment of inflammatory cells autodigestion of pancreas/blood vessels/tissue pseudocyst development symptoms/clinical findings: severe abdominal pain intensified by eating N/V, diarrhea (possibly steatorrhea) wegith loss 9...
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- Spring '09