F09studyques11_PesticideTox - NST110 Fall 2009 Pesticide...

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NST110 Fall 2009 Pesticide Toxicology Study Questions 1. Definitions a. hyperpolarization b. desensitization c. bradycardia d. atropine e. auxin f. aromatase 2. What is DDT's primary mechanism of action? How does this affect neurotransmission? How do the initial effects (tremors, jitters) lead to the final effects (paralysis)? 3. DDT has a secondary mode of action (at lower concentrations) that leads to endocrine disruption. What DDT metabolite is responsible for this effect? What endocrine system does it disrupt? How does this metabolite disrupt the normal functioning of this endocrine system? Be sure to include details on how this endocrine system can normally induce transcription. 4. Why is DDT bioaccumulative (e.g. what is its chemical property that leads to this- is it ionic? hydrophilic? lipophilic? amphipathic? polar? etc)? 5. What are some examples of organochlorines? How do they disrupt neurotransmission? How is this mechanism different from DDT (hint- different target)? How is this mechanism similar to
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This note was uploaded on 07/03/2010 for the course NST 110 taught by Professor Unknown during the Spring '04 term at Berkeley.

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F09studyques11_PesticideTox - NST110 Fall 2009 Pesticide...

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