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Sample midterm 2 2008 key

Sample midterm 2 2008 key - YOUR NAME Student ID MIDTERM 2...

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YOUR NAME__________________________ Student ID#_________________ 1 NPB 100 MIDTERM 2 SPRING, 2008 Your signature______________________ Please check this box if you do not want your exam returned in the bins in the hallway… Question Score Total 1 18 2 4 3 16 4 4 5 8 6 28 7 8 8 14 TOTAL 100
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YOUR NAME__________________________ Student ID#_________________ 2 Question 1. (2 points each, 18 points total) Here is a list of types of synaptic plasticity. Label each figure or statement below with one type of plasticity. Each answer can be used more than once or not at all. Post-tetanic Potentiation Depression Facilitation Homosynaptic Long Term Depression Heterosynaptic Long Term Depression Associative Long Term Depression Long Term Potentiation Associative Long Term Potentiation Augmentation 1a. facilitation 1b. depression 1c. Long Term Potentiation
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YOUR NAME__________________________ Student ID#_________________ 3 1d. Posttetanic potentiation 1e. Associative Long Term Potetniation 1f. Associative Long Term Depression
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YOUR NAME__________________________ Student ID#_________________ 4 1g. Heterosynaptic Long Term Depression Stimulation of one synapse causes a decreased postsynaptic response at another synapse. 1h. Depression This is caused by depletion of vesicles from the presynaptic terminal. 1i. Posttetanic Potentiation This is caused by the slow release of calcium from intracellular stores. Question 2 (4 points) In this circuit I excites F, and F and E inhibit each other. E has postinhibitory rebound, F does not. Draw the activity in the circuit following the 3 action potentials that are shown occurring in I. Question 3 (2 points each, 16 points total) Here is a list of neurotransmitters from lecture. For each statement fill in the blank with a neurotransmitter (each neurotransmitter may be used several times or not at all). Serotonin, GABA, norepinephrine, acetylcholine, nitric oxide, substance P, dopamine, glutamate 3a. Substance P is a peptide neurotransmitter involved in pain transmission.
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