Lec22 2010 Final - Lecture 22 Regulation of Gene expression Monday Bio 93 Fall 2010 Reading Chapter 18 pages 356-366 Supplements Self Quiz Ch19 1 2

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•Reading: Chapter 18, pages 356-366 •Supplements: Self Quiz Ch19: 1, 2, 4, 6, 8, 9, 10, 11. 14 •Web activity 19A, 19C, 19F Lecture 22: Regulation of Gene expression Fall 2010, Bio 93, O’Dowd and Warrior, UCI - Copyright: All rights reserved Monday 11/15/10 Bio 93 Fall 2010
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Why the Rough ER is Rough or Translating secreted and membrane proteins Fall 2010, Bio 93, O’Dowd and Warrior, UCI - Copyright: All rights reserved Study Tip: Review Lecture 5 Fig. 17.21
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Figure 17.23 The molecular basis of sickle-cell disease: a point mutation The change of a single nucleotide in the DNA’s template strand leads to the production of an abnormal protein In the DNA, the mutant template strand has an A where the wild-type template has a T. The mutant mRNA has a U instead of an A in one codon. The mutant (sickle-cell) hemoglobin has a valine (Val) instead of a glutamic acid (Glu). Mutant hemoglobin DNA Wild-type hemoglobin DNA mRNA mRNA Normal hemoglobin Sickle-cell hemoglobin Glu Val C T T
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This note was uploaded on 11/22/2010 for the course BIOLOGY SC Bio Sci 93 taught by Professor Dr.dianeo'dowdanddr.rahulwarrior during the Fall '10 term at UC Irvine.

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Lec22 2010 Final - Lecture 22 Regulation of Gene expression Monday Bio 93 Fall 2010 Reading Chapter 18 pages 356-366 Supplements Self Quiz Ch19 1 2

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