Cogs11 Final Review Questions

Cogs11 Final Review Questions - Cognitive Science - Final...

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Cognitive Science - Final Review Study Guide Fall 2010- Professor Mary Boyle Alzheimer’s Disease and Traumatic Brain Injury History of AD – how did that change the concept of mental illness? It was percieved that AD was a natural occurance, a senality in old age. Memories begin failing in one's 40's/ sometimes at 32. What can we learn from the families of Yarumal? The genetics of AD is sometimes, dominant genetic trait eFAD the siblings had a 50% chance of getting the disorder. What are the differences/similarities between early and late onset AD? Early -> genetics Late-> Discuss the three biomarkers for AD that were presented in class. How are the biomarkers used to evaluate success of a potential AD drug? physical changes-> amyloid beta markers What is TBI? Traumatic brain injury -> external forces damage the brain (traumatica injury ) (shaking up) How does TBI change your risk for AD? What were the studies
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discussed? The two have similar characteristics: such as neurofibilary tangles, amloyid plaques, acetycholine deficiency tau immunoreactivity Studies on military personal leave inconclusive results Studies on high school athletics showed that TBI incidencts increase What does a chronic TBI damaged brain look like? Cerebral concussions and hemorraghe ApoE4? What is it? How does it relate to AD risk? Apoliporotein E--> have a negative impact on neurological recovery, increasing the risk of AD increase in beta amyloid build up, increased risk for dementia Lifestyle choices and long term consequences? Lifestyle choice can increase the environmental factors that contribute to dementia, or alzhemier's disease Losing your Mind and The Qualia of Having a Stroke – Jill Bolte and Readings from Clues from a Damaged Brain Why study a damaged brain? To see how the corrected brain worsks, study the broken brain to see what parts of the brain are responsible for what Tumors – discuss the different types malignant ->
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benign -> covered encapsulated metastasizing -> origin from a different part in the body, and continues in the brain. Seizure disorders – causes? Who was Hughlings Jackson? temporary brain dysfunctions -> wild uncontrollable activity of the cerebral neurons strokes, or conditions that irritate the brain injury to the head, development after mints cerebral neurons--> excessive release of glutamate Damage to the hippocampus --> damaged based on how often the seizures occur Hughlings Jackson--> epilepsy --> Cerebrovascular accidents – risk profile? hemorrhagic v. obstructive;
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This note was uploaded on 12/30/2010 for the course COGS 11 taught by Professor Boyle during the Fall '08 term at UCSD.

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Cogs11 Final Review Questions - Cognitive Science - Final...

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