SNBAL 1 - patients. Healthcare facilities are crawling with...

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When the etymology of “lymphocyte apopsotis” is examined, we see that “lymph” means “relating to the lymphatic system”, “cyte” means “cell” and “apoptosis” means “programmed cell death”. Literally, lymphocyte apoptosis is the death of lymphatic (white blood) cells after a certain programmed series of events. Apoptosis in cells is normal, but too little apoptosis can result in cells becoming cancerous, while too much can lead to tissue damage. Immunosuppression in patients with sepsis is caused by excessive apoptosis. The disease causes the release of “proapoptotic” substances, which stimulate the lymphocytes to undergo more apoptosis than normal. The destruction of the lymphatic cells results in the weakening of the patient’s immune system. Eventually, the amount of destruction caused to the immune system leads to “immune paralysis”, usually just before the patient dies from sepsis. Susceptibility to nosocomial infections is to be expected amongst immunosuppressed
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Unformatted text preview: patients. Healthcare facilities are crawling with opportunistic infections, and immunosuppressed patients would be the first to acquire them. Fluid leak into the interstitial spaces is normal, and results from the filteration of plasma from blood flow through capillaries. Interstitial fluid is essential, as it provides a medium for components of the blood to filter into body cells. The fluid is usually drained by the lymphatic system or absorbed into the tissue cells/blood stream. As long as the fluid is not stagnant and does not build up, the patient will be healthy. If a patient is diagnosed with edema, his lymphatic system has malfunctioned; edema causes swelling in the affected area, and occurs when the interstitial fluid is not properly recycled. As long as the body systems are working properly, interstitial fluid leak is normal and does not have any negative consequences....
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This note was uploaded on 02/14/2011 for the course VTPP 434 taught by Professor Wasser during the Fall '10 term at Texas A&M.

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