Pain (1) - PAIN reading B L Chapter 7 pp 114-122 PAIN...

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PAIN reading- B & L Chapter 7, pp 114-122
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PAIN qualitatively different from other sensory systems variability in link between stimulus and response, strong affective or emotional component strong determinant of behavior Strong stimulus-no pain (EXAMPLE: ATHLETE INJURED IN GAME ) No stimulus-pain (EXAMPLE: CHRONIC PAIN SYNDROMES)
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CHRONIC PAIN SYNDROMES example : phantom limb pain 10% amputees (also from spinal cord transection) ALTERATION OF PROCESSING OF PAI N SIGNALS modulation of pain processing by emotional/cognitive state where, how does that happen? First, follow pain information from periphery to brain
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Transduction- what is the stimulus for pain? (extremes of temperature, pressure, cutting, etc.) tissue damage causes synthesis or release of chemical mediators which act on the endings of nociceptors (pain fibers) pain fibers are chemoreceptors hyperalgesia: increased sensitivity to pain following tissue damage, especially heat damage
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SEROTONIN, BRADYKININ, PROSTAGLANDIN, and by indirect pathway, HISTAMINE transmitter released by primary afferents is called Substance P (pain fibers are C fibers or Aδ fibers)
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