Ch 32-38 Homework

Ch 32-38 Homework - CindyRadford Chapter32NCLEXQuestions 1....

Info iconThis preview shows pages 1–4. Sign up to view the full content.

View Full Document Right Arrow Icon
Cindy Radford Chapter 32 NCLEX Questions 1. C 2. D 3. C 4. B 5. A 6. B 7. D 8. B 9. A 10. C 11. A Chapter 33 Critical Thinking Questions 1. Risk Factors For Hypertension: Age, Gender, Ethnicity, Family History, Smoking History, Alcohol Intake 2. Target Organ Damage:   ECG shows left ventricular hypertrophy, Serum Creatinine 1.6mg/d, Urinalysis 30mg/dl, Retinopathy. 3. Being a hyper  or laid back person doesn’t necessarily control your Blood Pressure 4. Risk for noncompliance related to side effects of medication. 5. Smoking increases risk for cardiovascular disease, checking BP regularly and keeping a journal. Chapter 33 NCLEX Questions 1. D 2. B 3. D 4. B 5. D 6. D 7. A
Background image of page 1

Info iconThis preview has intentionally blurred sections. Sign up to view the full version.

View Full DocumentRight Arrow Icon
8. D Chapter 34 Critical Thinking Questions 1. Left Common Carotid Artery   2. Atherosclerosis is the major cause of CAD. It is characterized by a focal deposit of cholesterol and lipids, primarily within the intimal wall of  the artery. The genesis of plaque formation is the result of complex interactions between the components of the blood and the elements  forming the vascular wall. Inflammation and endothelial injury play a central role in the development of atherosclerosis. Age, gender (men),  Ethnicity (white), and genetic predisposition are all Risk factors for CAD. Age, gender, and ethnicity are all risk factors that Mr. D already  had. 3. Angina, chest pain, is the clinical manifestation of reversible myocardial ischemia. Chronic Stable Angina is chest pain that occurs  intermittently over a long period with the same pattern of onset. MI pain is severe, immobilizing chest pain is not relieved by rest, position  change, or nitrate administration. 4. Sustained ischemia, causing irreversible myocardial cell death (necrosis). 5. Changes in ST segment are caused by ischemia and infarction. Cholesterol clogs the arteries.  6. O2 increased to provide for oxygen saturation, Aspirin ordered to break up clots, Nitro and morphine sulfate used to treat pain, Lopressor  ordered to decrease heart rate, Vital signs q10 minutes to monitor closely. 7. Impaired O2 exchange related to MI Chapter 34 NCLEX Questions 1. C 2. A 3. B 4. D 5. C 6. C 7. B 8. D Chapter 35 Critical Thinking Questions 1. It is a defect in the ability of the ventricles to contract (pump). The left ventricle loses the ability to generate enough pressure to eject blood  forward through the aorta. Over time the LV becomes thin-walled, dilated, and hypertrophied. The dyspnea is caused by increased pulmonary  pressures secondary to interstitial and alveolar edema. 
Background image of page 2
2. Dyspnea, Edema, Crackles, Murmurs, Increased Heart Rate.
Background image of page 3

Info iconThis preview has intentionally blurred sections. Sign up to view the full version.

View Full DocumentRight Arrow Icon
Image of page 4
This is the end of the preview. Sign up to access the rest of the document.

Page1 / 6

Ch 32-38 Homework - CindyRadford Chapter32NCLEXQuestions 1....

This preview shows document pages 1 - 4. Sign up to view the full document.

View Full Document Right Arrow Icon
Ask a homework question - tutors are online