Hemorrhage+Workseet+2011

Hemorrhage Workseet - indicated in box 2D 4 What is the mechanism behind changes in boxes 2G and 2H 3A(From 2C 3B 3D Arteriolar 3 Fluid shifts 3C

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Response to hemorrhage worksheet 1) Immediate Effects on Mean Arterial Pressure 1A 1B 1C 1D 1E 1F Blood volume EDV ) Short term responses to hypotension Cardiac output Magnitude: - 30 mm Hg MAP Sympathetic output to blood vessels 2) Short term responses to hypotension 2A (from 1F) 2B 2C MAP Baroreceptor activity 2D E 2F 2I Epinephrine levels Heart rate Sympathetic output to heart 2E 2G 2J Venous return 2H Magnitude: +18 mm Hg MAP Total peripheral resistance
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Questions, Parts 1) and 2): 1) What is the net change in MAP after blood loss? What is responsible for reducing the magnitude of the initial fall in MAP 2) Why is the change in venous return (box 2G) so important in raising MAP? 3) What is the neural pathway that produces the change in epinephrine levels
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Unformatted text preview: indicated in box 2D? 4) What is the mechanism behind changes in boxes 2G and 2H? 3A (From 2C) 3B 3D Arteriolar 3) Fluid shifts 3C Blood volume 3E 3F 3G lood P c Sympathetic output to blood vessels vascular resistance et filtration Blood volume Net filtration pressure Questions: 1) Consider the change indicated in box 3G. If the subject does not bleed anymore or consume fluid, where does the change in blood volume come from? 2) Will the change shown in 3G improve the problem of hypotension? How? 3) What will be the effect of the above fluid shift on hematocrit (% of blood consisting of cells)? 4) How do you think overall lymph flow will compare before and after hemorrhage?...
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This note was uploaded on 07/09/2011 for the course NPB 101 taught by Professor Fuller,charles/goldberg,jack during the Winter '08 term at UC Davis.

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Hemorrhage Workseet - indicated in box 2D 4 What is the mechanism behind changes in boxes 2G and 2H 3A(From 2C 3B 3D Arteriolar 3 Fluid shifts 3C

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