Anxiety and post - 1. Changes in brain function lead to low...

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Anxiety and post-traumatic stress disorder Aetiology GAD Some research suggests that GAD may run in families, and it may also grow worse during periods of stress. GAD usually begins at an early age and symptoms manifest themselves more slowly than in most other anxiety disorders. Some people with GAD report onset in early adulthood, usually in response to a life stressor. Once GAD develops it can become chronic Agoraphobia Both genetic and environmental factors appear to play a role Predisposition of responding to situations as dangerous maybe genetic and suggests an evolutionary role First degree relatives with agoraphobia is also a risk factor PTSD Caused by either a physical or psychological trauma, or more commonly both Genetics 1. Evidence of hereditary elements 2. Increased prevalence of PTSD in monozygotic twins Neuroendocrine
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Unformatted text preview: 1. Changes in brain function lead to low levels of cortisol and high levels of catecholamine’s in patients urine in relation to stress 2. In contrast unaffected individuals release have a lower catecholamine to cortisol ratio 3. Low cortisol levels may predispose an individual to PTSD • Risk factors 1. Military service 2. Traumatic experience in childhood 3. Foster care Important questions to ask a patient • How long have you felt like this? • What makes you anxious? • What feelings do you experience when anxious? • Do you ever have flash backs? • What brings the flashbacks on? • How do you cope with your anxiety? • Do you avoid things which make you anxious? • Do you ever notice your heart racing, shortness of breath, chest pain, dizziness? • Are you afraid of dying when you’re anxious?...
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This note was uploaded on 12/04/2011 for the course ANTHRO 2000 taught by Professor Monicaoyola during the Fall '10 term at Broward College.

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Anxiety and post - 1. Changes in brain function lead to low...

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