Ch 17 Bio - Chapter 17 Genome Sequencing, Molecular Biology...

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Chapt er 17, page 1 Chapter 17 Genome Sequencing, Molecular Biology and Medicine
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Chapt er 17, page 2 oncogenes: t umor suppressors: wild type receptor oncogenic form of receptor wild type receptor oncogenic form of receptor
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Chapt er 17, page 3 chronic myolegenous leukemia
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Chapt er 17, page 4 Sickle cell anemia 1-5% distribution of malaria by P falciparum HbS/ HbS HbA/ HbA 5-10% 1-5% 15-20% distribution of malaria by P falciparum 10-15%
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Chapt er 17, page 5 size 10 kb 5 kb 2 kb 1 kb 0.1 kb HbA: Pro - Glu - Glu CCT GAG GAG HbS: Pro - Val - Glu CCT GTG GAG MstI : CCTNAGG HbA: MstI MstI MstI HbA HbA HbA HbS HbS HbS HbA HbA HbA HbS HbS HbS 0.2 kb 1.1 kb
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Chapt er 17, page 6 CCTGAGGAG GGACTCCTC CCTGTGGAG GGACACCTC
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Chapt er 17, page 7
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Chapt er 17, page 8 Chapter 17 Key Words Genetic therapy Genetic screening Genome Proteome RFLP (Restriction fragment length polymorphism) VNTRs (variable number of tandem repeats) Tumor suppressor gene Oncogene
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Chapt er 17, page 9 Chapt er 17 Highlights: I. Cancer: A Disease of Genetic Changes A. Cancer cells differ from their normal counterparts B. Some cancers are caused by viruses C. Most cancers are caused by genetic mutations D. Two kinds of genes are changed in many cancers E. The pathway from normal cell to cancerous cell is complex II. Abnormal or Missing Proteins: The Mutant Phenotype
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Ch 17 Bio - Chapter 17 Genome Sequencing, Molecular Biology...

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