Handout #8 - Student Version

Handout #8 - Student Version - TA: Ishita Desai Handout #8...

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TA: Ishita Desai Handout #8 – Student Version GLP-1 and GIP are ________________ that STIMULATE/INHIBIT the secretion of insulin. What are the numerous effects of insulin? What stimulates and inhibits the release of glucagon? What are the not-so-numerous effects of glucagon? DIABETES What are the mechanisms for polydipsia and polyuria? ________________ is the third “P” of diabetes. This is because glucose is not metabolized signals hunger. What allows for HbA1c levels to be a good measure of BGL over the course of the past few months?
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TA: Ishita Desai Diabetic ketoacidosis is a serious problem for type I diabetics. The mechanism behind the problem is: - Glucose is not available as a form of energy (for brain!) increased fatty acid beta- oxidation -> increase ketoacids (acetoacetic and OH-butyric acids) dec blood pH metabolic acidosis (ketoacidosis) CNS depression (lose consciousness/coma), hyperventilate (until remove CO2), acetone breath o Tx with insulin and hydration (w/electrolytes)
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This note was uploaded on 04/06/2012 for the course BICD 150 taught by Professor Fortes during the Winter '09 term at UCSD.

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Handout #8 - Student Version - TA: Ishita Desai Handout #8...

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