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Ct features of primary sclerosing cholangitis

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Unformatted text preview: al infiltrating lesions grow along bile ducts. Intraductal tumors, polypoid or sessile. Extrahepatic cholangiocarcinoma, duct obstructing tumor Intrahepatic features? CT features of primary sclerosing cholangitis? appearance) with thickening of bile duct. Complications: obstruction, cholestasis, biliary cirrhosis, cholangiocarcinoma. Multiple segmental strictures (beaded Choledochal cystI,types? dilation of CBD. Type II, CBD diverticulum. Type III, choledochocele, protrudes into duodenal lumen. Type IVa, intrahepatic saccular dilatations. IVb, extrahepatic multiple cystic dilatations. Type V, Caroli's disease. Type cystic CT findings of acute cholecystitis? 75%, GB distension > 5 cm, GB wall thickening > 3 mm, Early-phase enhanced GB fossa, Pericholecystic stranding, Air in GB wall (emphysematous) Gallstones in gallbladder Gallbladder carcinoma CT features? Focal or diffuse wall thickening, Mass containing stones replaces GB and invades liver. Polypoid soft-tissue mass, CT features of lymphoma in abdomen Multiple enlarged nodes, Coalescence of enlarged nodes to form multinodular masses which may encase vessels, Conglomerate nodal masses CT features of AIDS in abdomen? lymphoma, Kaposi's), Focal, small low-density liver lesions (TB, Histo, lymphoma, Kaposi) Focal, small low density spleen lesions (MAI,Cocci, candida, PCP, lymphoma) Splenic or LN calcifications (PCP) Nephromegaly with striated nephrogram (HIV nephropathy) Kaposi's sarcoma (adenopathy, hepatosplenomegaly) AIDS-related lymphoma (any solid mass) Lymphadenopathy (MAI, Surgical indications for splenic trauma? Active bleeding, Large nonperfused portions Pseudoaneurysm formation Cortical rim sign? finding. Faint enhancement of kidney periphery in renal infarction. Renal capsule supplied by separate arteries Delayed Complications of pancreatic trauma? Pseudocyst formation hemorrhagic pancreatitis Abscess Fistula Shock bowel? Severe hypotension and hypoperfusion of bowel. Diffuse dilation of small bowel with wall thickening and increased wall enhancement Extraperitoneal bladder rupture? retropubic space, along abdominal wall, scrotum, thigh, and retroperitoneum. Contrast leakage into Intraperitoneal bladderparacolic gutters and surrounding bowel Contrast in rupture? Which adrenal gland more susceptible to traumatic injury? Right adrenal gland. compression of gland between liver and spine. Liver segment I? lobe, Ligamentum venosum and IVC separate it from liver Caudate Liver segments II and III? of left lobe. II-superior. III-inferior. Lateral division Liver segments IV? Medial division of left lobe. IVa-superior. IVb-inferior. Liver segments V and VIII? of right lobe. VIII-superior. V-inferior. Anterior segments Liver segments VI and VIII? of right lobe. VII-superior. VI-inferior. Posterior segments Third inflow? Areas of liver supplied by aberrant systemic veins. Porta hepatis, adjacent to gallbladder, adjacent to fissure of ligamentum teres. Increased liver attenuation? Amiodarone Hemochromatosis (secondary form-hemosiderosis, blood transfusions) Liver nodules in cirrhosis? Regenerative nodules Dysplastic nodules Small HCC nodules Metastatic disease Hemangiomas CT features of portal hypertension?vessels Enlarged portal vein, > 13 mm. Splenomegaly Ascites Portosystemic collateral CT features of Budd-Chiari syndrome? Enlarged caudate lobe. Central liver enhances early and peripheral liver enhances late. Clinically significant liver lesions? Hepatic adenoma Metastases Hepa...
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