O tissue venous pao2 20 mmhg or sao2 venous 40

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Unformatted text preview: s 40% - arterial PaO2 < 38 mmHg arterial or SaO2 < 70% or 70% Increase arterial PaO2 > 60 mmHg(SaO2 > 90%) or venous SaO2 > 60% or O2 dose either flow rate (L/min) or FiO 2 (%) Risks of Oxygen Therapy Risks O2 toxicity: - very high levels(>1000 mmHg) CNS toxicity and seizures - lower levels (FiO2 > 60%) and longer exposure: capillary damage, leak and pulmonary fibrosis capillary - PaO2 >150 can cause retrolental fibroplasia - FiO2 35 to 40% retrolental can be safely tolerated indefinitely can CO2 narcosis: - PaCO2 may increase severely to cause respiratory acidosis, somnolence and coma acidosis, - PaCO2 increase secondary to combination of PaCO a) abolition of hypoxic drive to breathe MECHANICAL VENTILATION MECHANICAL Non invasive with a mask Invasive with an endobronchial tube Invasive MV can be volume or pressure cycled MV For hypercapnia: - MV increases alveolar ventilation and lowers PaCO2, corrects pH PaCO corrects rests fatigues respiratory muscles For hypoxemia: For - O2 therapy alone does not correct hypoxemia caused by shunt caused - Most common cause of shunt is fluid filled or Most collapsed alveoli (Pulmonary edema) collapsed...
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This document was uploaded on 02/05/2014.

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