ESS 3091- 5- Exercise Metabolism- Student-2

ESS 3091- 5- Exercise Metabolism- Student-2 - Exercise...

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Exercise Metabolism
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Exercise Increase in cellular demand for ATP Rate of cellular work increases (metabolic rate) Skeletal muscle contraction Increased oxygen consumption (VO2)
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Steady State Exercise
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Recovery “Oxygen deficit” Steady-state exercise Oxygen debt Classic theory: oxygen debt repays oxygen deficit But…Oxygen debt >> Oxygen deficit Rapid portion (~5-20 min) Slow portion (>20 min-<24 hr)
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Recovery EPOC What: sustained elevated metabolic rate post-ex. Continued SNS stimulation of metabolism Epi, Norepi, thyroid hormone Elevated respiration, HR & body temperature Why: PCr replenishment, stored ATP, oxidative metabolism of lactate, glycogen replenishment, cellular O2 EPOC Duration Exercise volume (intensity x duration) Glycolytic substrate replenishment (post-ex.feeding)
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Intermittent Activity EPOC →↑caloric expenditure post exercise Intermittent bouts of exercise vs. single, sustained bout
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Graded Exercise Progressive, continued increase in exercise intensity Progressive transition from primarily oxidative to non-oxidative ATP production ↑lactate synthesis
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Graded Exercise VO2max (VO2peak) Oxidative capacity Max aerobic power CV fitness VO2peak
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Lactate As exercise intensity increases : Shift from oxidative to non-oxidative mechanisms Lactate production increases as fast glycolysis contributes progressively to ATP synthesis O2 availability (delivery, extraction) Recruitment of FG vs. FOG vs. SO fibers Capacity to shuttle NADH to mitochondria Build-up of NADH in cytoplasm: ↑ fast glycolysis
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Lactate Threshold = Lactate entry (production) >> Lactate removal (oxidation) The inflection point that suggests the shift towards predominantly fast glycolytic ATP synthesis Progressive ↑ in lactate prior to LT, exponential ↑ in lactate @ LT ~50% VO2max untrained ~75% VO2max trained
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Lactate Training= Delayed shift from oxidative to non- oxidative mechanisms greater oxidative capacity Greater efficiency of lactate shuttles (“lactate consumer”)
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Exercise Intensity & Fuel Selection As exercise intensity increases there is greater reliance on CHO and reduced reliance on FAT ↑ blood epinephrine levels SO → FOG → FG fiber recruitment Oxidative → Non-oxidative pathways ↓ O2 availability ↓ opportunity for oxidative mechanisms
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Exercise Intensity & Fuel Selection Respiratory exchange ratio (RER) = VCO2 / VO2 1.0= CHO 0.7= FAT
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RER
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RER > 1.0 w/ high intensity, NON- steady state exercise H+ from HLA, ↑ NADH + H+ production (↑ATP use) Bicarbonate buffering system: Expired at lungs= ↑CO2 part of RER VCO2/ VO2
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