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3.Generally, after neurotransmitters are released in the neuromuscular junction, many of the neurotransmitters or some of their components are recycled by a reuptake mechanism inherent in the presynaptic neuron. Considering that the reuptake mechanism is often a sodium-dependent process that utilizes membrane potential, what type of membrane transport might you expect to be in play here?The type of membrane that is expected to play a role in this is facilitated diffusion. Facilitated diffusion plays a role because substances are moving in and out of the cells and down into their concentrations gradient through the protein channels in the cell membrane. (Membrane transport 2016) In a reuptake reabsorption of a neurotransmitter by a neurotransmitter
A BOTCHED BOTOX PARTYtransporter of pre-synaptic neuron occurs after it has performed its function of transmitting of neural impulses.
A BOTCHED BOTOX PARTYCase Study Questions Part III1.You have isolated a long hydrophobic molecule that inserts itself within biological membranes. Specifically, it spans both layers of the lipid bilayer after insertion. Would you expect this molecule to mimic the effect that some general anesthetics have? Why or why not? Refer to the general anesthetic mechanism outlined in Figure 5 as a resource. 2. Compare and contrast local anesthetics, some general anesthetics, and botulinum neurotoxin in terms of the effect they have on neuronal excitability and function.Local and general anesthetics are used more often than botulinum neurotoxin simply because it causes pain and different medical conditions. This product affects neuronal excitability by causing wrinkling, lines and creases in the face, chin, and neck. General anesthetics aren’t exactly understood as much as the
A BOTCHED BOTOX PARTYothers listed in the chemical sense. Typically, general anesthetics potentiate the activation of inhibitory postsynaptic channels or inhibit the activation of excitatory synaptic channels. In other words, general anesthetics work to make neurons produces active action potential (Pudmed, 2006). Local anesthetics interact with sodium channels to reduce the excitability of nerve cells (Pubmed, 2006).3. Predict possible clinical outcomes of injecting botulinum neurotoxin intravenously, like a general anesthetic would be administered to a patient. Botulinum neurotoxin is said be painful when given to patients, and different types of medical conditions are present when this is given. It is also said to reduceabnormal contractions in the muscles to allow the muscles to become less stiff andrelax (Pubmed, 2006). If it was injected intravenously the chances of the outcome is 50/50. The predicted outcome is that many fatalities will occur in a short