10.Contrast the effects of aldosterone, ADH and atrial natriuretic peptide on the renal tubule. 11.Predict how ADH hypersecretion would affect sodium concentration of urine, and explain why. 12.Concisely contrast the role of the countercurrent multiplier with that of the countercurrent exchanger.13.How would the function of the collecting duct change, if the nephron loop did not exist OR a drug was administered that inhibited the function of the nephron loop? 14.Define oliguriaand polyuria. Which of these is characteristic of diabetes? 15.Identify a cause of glycosuria other than diabetes mellitus. 16.How is the diuresis produced by furosemide similar to the diuresis produced by diabetes mellitus? How are they different?
AP2-BIOL 2402 – From Saladin Textbook – ‘Before You Go On’ Study Guide F2015 For Exam #3 – Chapters 22, 23 and 24 17.Explain why GFR cannot be determined by measuring the amount of NaCl in the urine. Name a compound naturally found in human urine that is used clinically to estimate GFR? Why can that compound be used to determine GFR? Chapter 24 – Fluid/Electrolyte Balance and Acid-Base Balance These can also be found at the end of each section in the chapter. 1.List five routes of water loss. Which one accounts for the greatest loss? Which one is the most tightly regulated? 2.Explain why even a severely dehydrated person inevitably experiences further fluid loss.
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