Meld 378lnserum bilirubin mgdl 112lninr 957lnserum

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MELD = 3.78×LN[SERUM BILIRUBIN (MG/DL)] + 11.2×LN[INR] + 9.57×LN[SERUM CREATININE (MG/DL)] + 6.43 40 OR MORE — 71.3% MORTALITY 30–39 — 52.6% MORTALITY <9 — 1.9% MORTALITY 139
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Q- HEPATIC ENCEPH. WHICH OF THE FOLLOWING IS NOT A PRECIPITATING FACTOR FOR HEPATIC ENCEPHALOPATHY IN PATIENTS WITH CHRONIC LIVER DISEASE - (AIIMS MAY 05) A) HYPOKALEMIA B) HYPONATREMIA C) HYPOXIA D) METABOLIC ACIDOSIS 140
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THE KEY FACTOR ASSOCIATED WITH THE PATHOGENESIS OF HEPATIC ENCEPHALOPATHY IS INCREASE IN SERUM AMMONIA LEVELS. AMMONIA IS TOXIC TO THE NEURONS AND IS CONSIDERED TO BE PRIMARILY RESPONSIBLE FOR ENCEPHALOPATHY. 141
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WHICH OF THE FOLLOWING IS NOT A PRECIPITATING FACTOR FOR HEPATIC ENCEPHALOPATHY IN PATIENTS WITH CHRONIC LIVER DISEASE - (AIIMS MAY 05) A) HYPOKALEMIA B) HYPONATREMIA C) HYPOXIA D) METABOLIC ACIDOSIS D 146
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TRIPHASIC WAVEFORMS IN HEPATIC ENCEPHALOPATHY OCCURS IN STAGE OF - (NEET/DNB PATTERN) A) COMA STAGE-IV B) CONFUSION STAGE-I C) DEEP COMA STAGE-V D) DROWSINESS STAGE-II A 147
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A SYMMETRIC HIGH-VOLTAGE, TRIPHASIC SLOW WAVE PATTERN IS SEEN ON EEG IN THE FOLLOWING A) HEPATIC ENCEPHALOPATHY (AIIMS MAY 06) B) UREMIC ENCEPHALOPATHY C) HYPOXIC ENCEPHALOPATHY D) HYPERCARBIC ENCEPHALOPATHY A 148
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WHILE THE TYPICAL TRIPHASIC WAVES ARE COMMON IN HEPATIC ENCEPHALOPATHY . ATYPICAL TRIPHASIC WAVES OCCUR IN OTHER METABOLIC ENCEPHALOPATHIES SUCH AS – UREMIA HYPOTHYROIDISM HYPERCALCEMIA HYPONATREMIA • HYPOGLYCEMIA LITHIUM INTOXICATION 149
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VIRAL HEPATITIS ALMOST ALL CASES OF ACUTE VIRAL HEPATITIS ARE CAUSED BY ONE OF FIVE VIRAL AGENTS: HEPATITIS A VIRUS (HAV), HEPATITIS B VIRUS (HBV), HEPATITIS C VIRUS (HCV), HBV-ASSOCIATED DELTA AGENT OR HEPATITIS D VIRUS (HDV), AND HEPATITIS E VIRUS (HEV). OTHER TRANSFUSION-TRANSMITTED AGENTS (E.G., "HEPATITIS G" VIRUS AND "TT" VIRUS, HAVE BEEN IDENTIFIED BUT DO NOT CAUSE HEPATITIS). ALL THESE HUMAN HEPATITIS VIRUSES ARE RNA VIRUSES, EXCEPT FOR HEPATITIS B, WHICH IS A DNA VIRUS 151
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THE LIVER BIOPSY IN ACUTE HEPATITIS DUE TO HEPATITIS B VIRUS IS LIKELY TO SHOW ALL OF THE FOLLOWING, EXCEPT- A) BALLOONING CHANGE OF HEPATOCYTES B) GROUND GLASS HEPATOCYTES (AIIMS MAY 04) C) FOCAL OR SPOTTY NECROSIS D) ACIDOPHIL BODIES 153
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PATHOLOGICAL FEATURES OF ACUTE VIRAL HEPATITIS :- 1. PANLOBULAR INFILTRATION OF MONONUCLEAR CELLS 2. HEPATIC CELL DAMAGE - IT CONSITS OF :- A) HEPATIC CELL NECROSIS - FOCAL OR CENTRIZONAL. B) BALLOONING OF CELLS - DIFFUSE SWELLING C) ACIDOPHILIC DEGENERATION OF HEPATOCYTES - COUNCILMAN BODIES D) CELL DROPOUT 154
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3. HYPERPLASIA OF KUPFFER CELLS 4. VARIABLE DEGREE OF CHOLESTASIS 5. HEPATIC CELL REGENERATION - ROSETTE OR PSEUDOACINAR FORMATION AND MULTI NUCLEATED CELLS. 6. LOBULAR DISARRAY - LOSS OF NORMAL ARCHITECTURE.
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  • Winter '16
  • jean grey
  • chronic hepatitis, chronic liver failure

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