Healthy child after all congenital syphilis

healthy child after all.
Congenital syphilis classification
I. Syphilis of the fetus and placenta;
II. Early congenital syphilis (a child under two):
1) Nurseling;
2) Infancy.
III. Late congenital syphilis (a child after two).
Placental syphilis — Treponema pallidum, penetrating into the placenta, forms its
anatomico-morphological changes manifested in marked villi sclerosis, epithelium
degeneration, cell infiltration of the vessels walls, edema and increase in placental weight
(normal placental/ fetal weight ratio is 1:6; after syphilitic infection-1:3).
Fetal syphilis — Affection of internal organs manifests itself in their increase and induration
because of diffuse inflammatory infiltrate development and further excrescence of connective
tissue (especially in the liver and spleen). Phenomena of so called "white pneumonia" because
of specific infiltration of interalveolar septums, hyperplasia and desquamation of alveolar
epithelium occur. A stillborn skin is macerated and erosive in some places. Epidermis is loose
because of the retarded hypodermal cell development. Skin folds easily. It is loose and
wrinkled (develops a senile appearance).
Clinical syphilis manifestations in born alive newborns can be observed at birth or
sometimes they may develop first two years (early congenital syphilis). Skin affection at the
early congenital syphilis can be divided into two groups:
I. All types of typical secondary syphilids (mainly papular ones);
II. Special forms:
1) Pemphigus syphiliticus;
2) A diffuse papular infiltration of skin (Hochsinger);
3) Syphilitic rhinitis.

Pemphigus syphiliticus — It either exists or manifests itself the first days of newborn life.
Eruptions are tense vesicles filled with serous or se-ropurulent (infrequently hemorrhagic)
fluid localizing on the infiltrated floor and without any signs of acute inflammation. It is
surrounded by reddish-brown border without tendency to confluence. Its fluid contains
Treponemas pallidum.Vesicles appear first on palms and soles and only then on other parts of
skin. It is differentiated from epidemic impetigo neonatorum. Infectious dermatosis develops
first 5-10 days after birth. Vesicles are localized everywhere. They are flaccid with signs of
inflammation and have a tendency to peripheral growth and confluence. Signs of intoxication
are typical.
A diffuse papular infiltration of skin (Hochsinger) develops first 3 months of newborn life.
It is localized on palms, soles, buttocks, chin, lips, and forehead. Skin in the places of affection
is infiltrated, purple-red with smooth lustrous surface. Injury of skin leads to fissures in the
mouth corners. They form radial cicatrices of Robinson-Fourniet. Infiltration of palms and
soles is accompanied by maceration, skin folding, and scaling ("a hand of laundress").
Syphilitic rhinitis — It is the most typical affection of mucous membranes in the early
congenital syphilis. It develops in the first 3 -6 weeks of newborn life. It has three stages:
1.