Treatment of underlying cause and supportive

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 treatment of underlying cause and supportive respiratory therapy Infant respiratory distress syndrome : caused by inspiratory stridor (the sound showing something isn’t open) Infant Respiratory Distress Syndrome Usually related to premature birth Caused by a lack of surfactant in the alveoli Poorly developed alveoli are difficult to inflate: this causes atelectasis, and  decreased pulmonary blood flow- (causing pulmonary vasoconstrictions  and resulting in severe hypoxia) Poor lung perfusions and lack of surfactant 1. Increased alveolar capillary permeability 2. Fluid and protein are leaking into the interstitial area and alveoli- hyaline  membrane formation Signs & Symptoms Respiratory difficulties may be evident at birth or shortly thereafter Respirations rapid and shallow with chest retractions and flaring of the  bares Frothy septum and expiratory grunt Blood pressure falls Cyanosis and peripheral edema Severe hypoxemia and decreased responsiveness
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Irregular respirations with periods of apnea Decreased breath sounds Diagnostic tests:  arterial blood gas analysis   Treatment:  Glucocorticoids to women in premature labor, synthetic surfactant to  high-risk neonates, ventilation using CPAP, oxygen therapy and nitrous oxide  drugs Cleft Palate - Patients with cleft palate would have trouble feeding because they have a hole in the palate so there for can’t masticate (chew) or suck with straws/breastfeeding Chapter 20: Digestive System Disorders Children that have pyloric stenosis - they start projectile vomiting after they eat it is without nausea – they give them thickened food because it makes it heavier and if that doesn’t work they will use a balloon to stretch it or surgery Pyloric Stenosis : Narrowing and obstruction of pyloric sphincter occurs when muscle fibers thickens and narrow the outlet -Developmental anomaly may occur -Signs appear within several weeks after birth Nonbilious projectile vomiting after feeding Firm mass can be palpated at pylorus Infant fails to gain weight, dehydration, and hunger -Surgery required to remove obstruction Signs that may be acquired later in life Persistent feeling of fullness Increased incidence of vomiting Peptic/Gastric/Duodenal Ulcers -Most are due to H. pylori infection -Occur most commonly in the proximal duodenum (duodenal ulcers) -Also found in the antrum of the stomach (gastric ulcers) -Development begins with the breakdown of mucosal barrier Decreased mucosal defense Increased acid secretions predominant factor in duodenal ulcers -Damage to mucosal barrier predisposes to development of ulcers and is associated with Inadequate blood supply Excessive glucocorticoids secretion or medication Ulcerogenic substances (ASA, NSAIDs, Alcohol) Atrophy of gastric mucosa (chronic gastritis) - Increased acid-pepsin secretions Complications with peptic ulcer Hemorrhage: due to erosion of blood vessels Perforation : ulcer erodes through the wall Obstruction:
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