cause hyperplasia increased adhesion macrophage engulf LDL foam cells fatty

Cause hyperplasia increased adhesion macrophage

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cause hyperplasia, increased adhesion, macrophage engulf LDL-foam cells, fatty streak- triggers more immune/inflam response, ongoing progressive damage to vessel, produces collagen to form plaque, calcify and occlude flow, can rupture and cause ischemia or infarct
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after eating, PVD, renal artery stenosis Peripheral Arterial Disease Age, more common in AA, smoking DM Pain with ambulation- intermittent claudication, Athlerosclerotic changes which affect extremities; decreased perfusion gradually, Coronary Artery Disease and Acute Coronary Syndrome 2. Examine the risk factors, etiology, clinical manifestations, pathophysiology and consequences of coronary artery disease and acute coronary syndrome. CAD- advanced age, male, post menopausal women, obesity, family hx, HTN, dyslipidemia, smoking, DM, sedentary lifestyle, high fat diet, a.Explain how modifiable risk factors contribute to the development of coronary artery disease:
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released during MI—vasoconstriction, increase afterload, myocardial workload, necrosis in 20 min, coagulative necrosis, area determines fxn of heart and CMd.Describe those persons most at risk for, the etiology of, and the presentation (clinical manifestations) of stable, prinzmetal angina and silent ischemia. TypePersons at Risk Etiology Clinical ManifestationsStable AnginaWomen, elderly, DM, CAD causesmyocardial ischemia, CP relieved with restPrinzmetal AnginaDecreased vagal activity, hyperactive sympathetic nervous system,decreased nitricoxide, altered Ca channel fxnCP at rest, at night caused by
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  • Fall '15
  • david,mary

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