such as Diabetes mellitus, and medications such as beta blockers (Arcangelo, Peterson, Wilbur & Reinhold, 2017). The behavior has a tremendous impact on both HLD and HTN. There is the exposure of individuals to diet high in salts and cholesterol daily due to the consumption of fast foods in addition to physical inactivity. As a result, modification of dietary intake, physical activity, and stress reduction prevent the occurrence of HTN and HLD (Oikonomou et al., 2018). Diet and nutrition affect the metabolism and response of drugs within the body. Food plays a role in the pharmacokinetics as food can either decrease or increase the absorption of a drug, thereby affecting the bioavailability of the medication. For example, juices such as apple, orange, and grape alter the absorption of atenolol by the body, reducing its effect. The goal in management and treatment of HTN is reducing cardiovascular mortality and morbidity as well as reduction of renal issues. The first line of therapy for stage I HTN is the use of hydrochlorothiazide which is a thiazide diuretic, and there can be the addition of ACE inhibitors, beta blockers, calcium channel blocker or angiotensin II receptor blocker. The above patient AO is on beta-blocker atenolol. For stage II HTN the therapy is the same as for stage I but with the addition of vasodilator such as hydralazine especially for patients with essential HTN.