GENERALLY LAST NO MORE THAN AN HOUR AND COMPLETELY RESOLVE WITHOUT RESIDUAL

Generally last no more than an hour and completely

This preview shows page 23 - 30 out of 124 pages.

GENERALLY LAST NO MORE THAN AN HOUR AND COMPLETELY RESOLVE WITHOUT RESIDUAL DEFICITS THOROUGH HISTORY OF EVENT IS ESSENTIAL, HOW IT BEGAN, SYMPTOMS EXPERIENCED, HOW LONG IT LASTED IF CAROTID OBSTRUCTION IS SUSPECTED, CAROTID DUPLEX ULTRASOUND STUDIES ARE DONE TO DETERMINE IF OBSTRUCTION IN CAROTID ARTERIES IS PREVENTING NORMAL BLOOD FLOW FROM REACHING THE BRAIN
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TRANSIENT ISCHEMIC ATTACK (TIA) BLOOD TESTS, MRI, EEG IF THERE IS NEAR TOTAL OCCLUSION OF CAROTID ARTERY, EITHER AN ANGIOPLASTY PROCEDURE WITH STENT IMPLANTATION OR A CAROTID ENDARTERECTOMY IS CONSIDERED IF OCCLUSION FROM PLAQUE IS LESS THAN 60%, MEDICAL TREATMENT WITH DIET AND LIFESTYLE MODIFICATIONS AND MEDS TO PREVENT PLATELET AGGREGATION- ASPIRIN, CLOPIDROGEL, DIPYRIDAMOLE
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CEREBROVASCULAR ATTACK (CVA) STROKE, BRAIN ATTACK RISK FACTORS MODIFIABLE CIGARETTE SMOKING, COCAINE OR OTHER RECREATIONAL DRUGS DRINKING MORE THAN 2 DRINKS PER DAY HEART DISEASE (ESPECIALLY A-FIB) DM, HTN, HIGH CHOLESTEROL SEDENTARY LIFESTYLE TIA’S, HIGH RBC’S, ORAL CONTRACEPTIVES OR HRT NONMODIFIABLE AGE >65, HEREDITY ASYMPTOMATIC CAROTID BRUIT PRIOR STROKE AFRICAN AMERICANS, MALES ATHEROSCLEROSIS
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CEREBROVASCULAR ATTACK (CVA) STROKE, BRAIN ATTACK RISK FACTORS CULTURAL CONSIDERATIONS- AFRICAN AMERICANS HAVE ABOUT A 60% GREATER INCIDENCE OF STROKE THAN WHITES. HISPANICS HAVE A FAR GREATER RISK THAN NON-HISPANICS. UNTREATED HTN MAY BE THE RISK FACTOR INVOLVED
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CEREBROVASCULAR ATTACK (CVA) STROKE, BRAIN ATTACK PATHOPHYSIOLOGY CVA IS THE RESULT OF AN INTERRUPTION OF BLOOD FLOW TO A SPECIFIC AREA OF THE BRAIN (CEREBRAL ISCHEMIA) ISCHEMIA OF CELLS DIRECTLY CAUSES CELLULAR NECROSIS AND INFARCT ISCHEMIA CAN BE CAUSED BY CEREBRAL THROMBOSIS EMBOLUS INTRACEREBRAL HEMORRHAGE PRESSURE ON A BLOOD VESSEL
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CEREBROVASCULAR ATTACK (CVA) STROKE, BRAIN ATTACK RISK FACTORS HEALTH PROMOTION- DANGERS OF COCAINE OR METHAMPHETAMINE USE- BOTH CAUSE VASOCONSTRICTION AND BRAIN ISCHEMIA. COCAINE MAY ALSO CAUSE HEMORRHAGE. BOTH CAUSE A 5 FOLD INCREASE IN THE INCIDENCE OF STROKE CAROTID ARTERIES SUPPLY A MAJOR PORTION OF THE BLOOD THAT GOES TO THE BRAIN IF PLAQUE FORMS IN THESE ARTERIES AS A RESULT OF ATHEROSCLEROSIS, THE PERSON IS AT RISK FOR A STROKE AS BLOOD SUPPLY TO THE BRAIN IS DIMINISHED OR STOPPED LESS COMMON CAUSES- ARTERIAL SPASMS, COMPRESSION OF CEREBRAL VESSELS BY A TUMOR, LOCAL EDEMA, RUPTURE OF A CEREBRAL ANEURYSM OR ANOTHER DISORDER
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CEREBRAL ANEURYSM AND ARTERIOVENOUS MALFORMATION STRUCTURES THAT CAN CAUSE AN INTRACEREBRAL HEMORRHAGE ARE AN ANEURYSM AND AN ARTERIOVENOUS MALFORMATION ANEURYSM- ABNORMAL BALLOONING OF AN ARTERY WALL. MAY BE CONGENITAL OR CAUSED BY A WEAKENING OF THE ARTERY WALL FROM CHRONIC HYPERTENSION RUPTURE OF A BRAIN ANEURYSM CAUSES BLEEDING INTO THE SUBARACHNOID SPACE OR INTO THE VENTRICLES AN ARTERIOVENOUS MALFORMATION IS A CONGENITAL ABNORMALITY AND IS A TANGLED MASS OF MALFORMED, THIN-WALLED, DILATED VESSELS THAT FORM AN ABNORMAL COMMUNICATION BETWEEN THE ARTERIAL AND VENOUS SYSTEMS
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