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the condition to manageable levels. This is one study that has embodied two approaches that seems to help diabetic patients in dealing with their condition. Nonetheless, there is crucial information that could be missing from this research regarding the age, number of years sincediagnosis of type II diabetes, functionality of the kidneys and whether patients are under other intervention methods. Such information is crucial in such a study since it helps to distinguish the type of patient who would benefit from a particular method and those who would not benefit from the method. Nonetheless, this is a reliable study since it has relied on more than ten participants thus the findings are more reliable. On the other hand, Petersen, Dufour, Befroy, Lehrke, Hendler & Shulman (2005), preferred to conduct a research using eight participants whereby they investigated the mechanism by which weight loss improves insulin and basal stimulated rates of glucose metabolism among T2D patients. The intramyocellular lipid (IMCL) and intrahepatic lipid (IHL) contents were accessed using H magnetic resonance spectroscopy. The main target were obese participants. Consequently, then, the study utilized a moderately hypocaloric very-low-fat diet (3%) throughout the study. Notably, the lean control subjects were different from the diabetic patients in the sense that they were not insulin resistant in both liver and muscle. Some of the results obtained included a decrease in weight by 8 kg after a normalization of fasting plasma glucose concentrations. The overall data obtained from the study supported the hypotheses that moderate weight loss normalizes fasting hyperglycemia
REVERSAL OF TYPE II DIABETES 8for all those people diagnosed with poorly controlled T2D by mobilizing a relatively small pool of IHL. Still, the intervention technique is significant in normalizing the rates of glucose production in the body (Petersen et al., 2005). This study would only be beneficial for those type II diabetic patients who are obese. The mechanisms used would not necessarilyhelp the patients who are not interested in weight loss. As such, the study does not give a general intervention mechanism for all type of patients who are diagnosed with type II diabetes. Still, there is a limitation on the number of participants used for this study. Eight participants may not help to give a lot of varying information especially on other issues that would help in further investigation and give new information on the etiology and pathophysiology of those conditions. Most of the mechanisms that have been discussed so faronly include medication, exercise and balanced diet to help reverse type II diabetes. Nonetheless, there are measures that could be used to help patients particularly those suffering from obesity and T2D. Bariatric surgery can be used to give major improvements inglycemic control as well as substantial, sustained weight loss among type II patients with severe obese cases (Dixon, Le Roux, Rubino & Zimmet, 2012). Mingrone, Panunzi, De