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Polyuria b oliguria c disuria d anuria e hypostenuria

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polyuriab.oliguriac.disuriad.anuriae.hypostenuria377.Patient М., 16 years, was delivered to the hospitalin the extremely shock condition after car accident. There areplural fractures of both legs. ABP - 80/60 mmHg. Dailydiuresis 60-80 ml. What is the mechanism of decreased diuresisat this patient?a.decreased hydrostatic pressure in capillaries ofglomerulesb.increased colloid-osmotic pressure in capillaries ofglomerulesc.increased pressure of primary urine in Bowman’scapsuled.increased concentration of vasopressin in bloode.traumatic violation of urinary bladder innervation378.Patient М., 16 years, delivered in the hospital in theshock condition. ABP - 80/60 mmHg. Daily diuresis 60-80 ml.
Urine analysis: protein - 0,66 g/L, urine specific gravity 1,029.Residual blood nitrogen - 120 mmol/L; blood urea - 35mmol/L. What is the mechanism of azotemia in the patient?a.decreased glomerular filtration rate in kidneysb.violation of urea secretion in kidneysc.violation of protein metabolism in liverd.increased catabolism of proteins in the damagedtissuese.increased reabsorption in kidneys379.Patient М., 16 years, with craniocerebral traumawas delivered to the hospital in the shock condition. ABP -80/60 mmHg. Excretes 60-80 ml of urine for a day. In urine:protein - 0,66 g/L, urine specific gravity 1,029. What is themost possible cause of development of proteinuria at patient?a.ischemic affection of kidneysb.traumatic affection of kidneysc.inflammatory affection of kidneysd.inflammatory affection of urinary bladdere.traumatic affection of urinary bladder380.In the analysis of urine of patient K., 3 years, isglycosuria, diurnal excretion of glucose with urine is 1,2 g,degree of glycosuria is the same in day and night portions ofurine; glucose of blood - 3 mmol/L, glycemic line at sugarloading or injection of insulin is normal. What is the cause ofglycosuria at patient?a.excess of glucose in blood concentrationb.increased filtration of glucose in kidneysc.deficiency of enzyme glucose transport systems inthe tubulesd.high income of glucose with foode.violation of glucose consumption by tissues381.Patient К., 3 years, complaints about quickfatiguability, constant hunger, high thirst. There are noobjective changes in the internal organs. Urine analysis: urinespecific gravity 1,020 - 1,038 at daily diuresis 3,0 l. Diurnalexcretion of glucose with urine is 1,2 g. What pathogeneticmechanism caused changes of diurnal diuresis at patient?a.increased hydrostatic pressure in capillaries ofglomerulesb.decreased colloid-osmotic pressure in capillaries ofglomerulesc.decreased hydrostatic pressure in Bowman capsuled.increased osmotic pressure of urinee.decreasedsecretion of vasopressin in blood382.Patient A., 38 years, at 3-d year of disease ofsystemic lupus erythematosus it is revealed diffuse affection ofkidneys, with massive edema, expressed proteinuria. What hascaused the development of proteinuria at a patient?

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