1 Analyze how normal physiology is altered in liver disease.
dual blood supply consisting of a venous (portal) supply through the hepatic portal
vein and an arterial supply through the hepatic artery.
The venous outflow from the liver is carried by the va
PAT20 Week 11 Objectives : COPD
1. Discuss the etiology of Chronic Obstructive Pulmonary Disease (COPD)
COPD is a respiratory disorder caused largely by smoking and characterized by
progressive, partially reversible airflow obstruction, systemic manifesta
WEEK 8LIVER ch 46
Liver functions: metabolizes carbohydrates, lipids, and proteins, produces bile and detoxifies
endogenous and exogenous substances (hormones and drugs)
Gallbladder: stores and concentrates bile
Pancreas: regulates blood glucose by secret
PAT20A WEEK 1:
PATHOTHERAPEUTICS AND PHARMACOLOGY
1. Discuss drug classification.
Drugs are classified by chemical structure of active ingredient or by
pharmacological action (function).
Throughout the process of development, a drug will a
PAT20A Week 6/7: Stroke
Circulation System of the head and neck
1. Identify modifiable stroke risk factors.
Modifiable risk factors of stroke are those that can potentially be altered through lifestyle changes and medical
o 5 mill
WEEK 2 ACID-BASE BALANCE
Normal: 285-295 mmol/kg
fluid excess-too much water, too little solute< 285-295 < too much solute or too little water-fluid deficit
Types of IV fluids
Crystalloidssolutions contain small molecules, easily cross cell mem
Coronary Heart Disease & Acute Coronary Syndrome
Pathophysiology processes related to the physiological changes to the vascular
system associated with the HT, atherosclerosis and stroke.
*See week 6 and 7 notes Atherosclerosis and Stroke
PAT20 WEEK 6 ACUTE KIDNEY INJURY AND CHRONIC KIDNEY DISEASE
ACUTE KIDNEY INJURY (AKI)
an abrupt decline in kidney functionleads to a rise in serum creatinine or a reduction in urine
output (or both)
severity may be mild (little intervention) to severe (
[Fall - Week 1 - PAT20A
Introduction of Pathotherapeutics and Pharmacology
1. Discuss drug classification.
Drugs are classified by chemical structure of active ingredient or by pharmacological action (function).
Throughout the process of development, a dr
Week 4 Learning Objectives
1. Discuss the etiology of HT.
Hypertension (HTN) is sustained elevation of systemic arterial blood pressure (BP) and is
the leading cause for visits to primary care physicians. High BP is the most significant
FLUID VOLUME DEFICIT
Insufficient fluid intake
Goal: restore fluid volume, replace electrolytes,
eliminate the cause
Water loss = electrolyte loss
most common, results in decreased
circulating blood volume a
Unpleasant sensory and emotional experience associated with actual or potential tissue damage, and with
many diseases or health conditions.
Acute pain sudden and usually subsides when treated e.g. a headache
Persistent pain long lasting or
1. Non-steroidal anti-inflammatory drugs (NSAIDs)
MECHANISM OF ACTION AND DRUG EFFECTS
o Act through the inhibition of the LT pathway, the PG pathway, or both.
o Relieves pain, headache, and inflammation by blocking the chemical activity of either or bot